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Inhibition of GSH depletion by either high extracellular GSH or NAC may prevent increased ROS formation and control abnormal apoptosis as well as several other abnormal immune responses, cytokine as well as chemokine production in SLE patients

Because this phosphorylation cluster resides within the first 120 amino acids, we anticipated that N-terminal tagging would preferentially biotinylate key regulators coordinating with the hinge region

doi: 1 10.1016/j.jtcme.2014.1011.1036

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