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Pretreatment of HaCaT cells with an IP3R inhibitor indicates that GSK3 may regulate the activation of IP3R, and thereby modulate the release of Ca 2+ from the endoplasmic reticulum into the cytoplasm, thus indirectly regulating mitochondrial damage, and apoptosis

Lund ML, Sorrentino G, Egerod KL, Kroone C, Mortensen B, Knop FK, et al

The body therefore tends to preserve fat stores while trimming unneeded muscle mass unless regular exercise signals that muscle remains necessary and adequate protein intake supports its maintenance