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There has been heightened interest in the last few years regarding genetics and ones genetic predisposition to certain acquired diseases and illnesses

(B) Semaglutide, as a GLP-1 analog, can directly act on fat cells, increase the expression of thermogenic genes for browning phenotype maintenance, improve the expression of uncoupling protein 1 (UCP1), mitochondrial biosynthesis, and thermogenic marker, or promote fatty browning through nitric oxide synthase (NOS), neuropeptides (NPs), sirtuin 1 (STRT1), signal transducer and activator of transcription 3 (STAT3), and bone morphogenetic protein 4 (BMP4) signaling

Long P, Guo C, Wen T, et al

United States Department of Health and Human Services, Centers for Disease Control and Prevention and National Cancer Institute