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Grx2 knockout or overexpression exacerbates or diminishes lipid peroxidation in human lens epithelial cells or opacity in mouse lenses, respectively, after treatment with ferroptosis inducers or UV-B via modulation of mitochondrial structure and the GSH/GSSG ratio [128]
Observed molecular effects are dependent on experimental design, concentration, and model system, and are used to explore signalling mechanisms rather than defined biological or physiological outcomes
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These enzymes are considered to play a primary role in removing cells identified as damaged or dysfunctional within an organism
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