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ERK Activation Has an Essential Role in Liraglutide-Induced Cell Proliferation of PSCs ERK activation in PSCs is important in stimulating the growth effects of numerous stimuli

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Alanine mutations of E124 3.33 and R283 6.55 nearly abolished the binding of ghrelin-A2 and the ghrelin-induced receptor activity, indicating the critical role of the salt bridge in maintaining the integrity of the bifurcated binding pockets, and thus affecting ghrelin binding and receptor activation (Supplementary Figs

Li et al., 2015)

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