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However, after irradiation, GPx1-KO and wild-type mice exhibited almost similar levels of intestinal GPx activity, and this was due to the radiation-induced compensatory upregulation of GPx2 in GPx1-KO mice

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More recent studies have reported alterations in monoamine metabolism in the cerebrospinal fluid and plasma of individuals with ME/CFS, particularly elevated levels of the serotonin metabolite 5-hydroxyindoleacetic acid (5-HIAA), suggesting increased central serotonergic turnover (Demitrack et al., 1992

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