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Elevated IGF-1 LR3 activates growth signalling independently of GH receptor pathways, and the exogenous peptide does not participate in GH axis feedback loops

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Many patients in the trial were also receiving standard-of-care therapies for their underlying cardiovascular disease, with 90.1% of patients taking lipid-lowering medications, 86.2% antiplatelets, 70.2% beta-blockers, 45.0% ACE inhibitors and 29.5% ARBs

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