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In the MTX group, severe vacuolar degeneration and pyknotic nucleus in hepatocytes, increased number of activated Kupffer cells, inflammatory cell infiltration and marked sinusoidal dilatation and congestion were prominent features of the morphologic damage (Fig

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Future research should focus on optimizing ferritin's biocompatibility and enhancing its binding affinity to Gln antagonists through advanced molecular docking simulations and structural engineering techniques