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That flat selectivity profile is precisely what keeps MT2 useful in vitro: it serves as the positive-control full agonist against which subtype-selective ligands are benchmarked, and it is the parent scaffold for a large derivative family generated by single substitutions at positions 6, 7 and 9, including the MC3R/MC4R antagonist SHU-9119

MSSM-002 treatment eliminated clinical indicators of allergic asthma, including airway hyperreactivity and the total number of cells and percentage of eosinophils in the BALF [93]