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Activation of the P2XR7 receptor of hCMEC/D3 cells by ATP induced IL1 secretion, activated MMP9, and decreased ZO-1 and occludin expression (Yang et al., 2016)

A specific mechanism by which glutathione thiol and glutathione disulfide operate to this aim is the reversible thiol(ate)-exchange mechanism, through which an excess of glutathione thiol regulates in an inhibitory sense the biological pathways which produce reducing cofactors or conditions by binding to sensing, conformation-determining disulfide bonds of proteins in a process termed reversible protein glutathionylation [134]

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