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After describing UA biology, we summarized recent evidence on the association between UA and CVD by analyzing recent epidemiological, clinical, experimental, genetic (Mendelian randomization) and intervention studies with UA-lowering therapy

this effect occurs through the modulation of protein tyrosine phosphatase activity in IGF-1 signaling

258 Additionally, TREM2 has emerged as a potential therapeutic target due to its potential role in early AD in modulating neuroinflammation, A plaque deposition, and cognitive abilities

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