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adult population suffering from obesity, one might think that is a drop in the bucket

The contrasting responses to combined mTORC1 inhibition and ferroptosis induction in different cellular contexts are likely dictated by complex factors including genetic mutation landscape, the ratio of polyunsaturated to monounsaturated phospholipids, iron availability, the basal activities of the PI3K/mTORC1 pathway, and the overall cellular redox state

NS, nonsignificant.

77 This complex picture has been nicely reviewed by Connell and Venkataraman 78 and is summarized in Figure 5

In vitro, OGD-induced ferroptosis was ameliorated by GSH, which restored cell proliferation, reduced oxidative stress, and upregulated ferroptosis defense factors