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In Ringer solution, when the hematocrit was adjusted to 4%, PS exposure was not significantly different, although it was around half the level of PS exposure seen in the 0.4% hematocrit condition

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That's where GLP-1 receptor agonist therapy changes the equation

In this sense, several diseases or conditions or genetic disorders that are predisposing to eryptosis, including deficiency in glucose-6-phosphate dehydrogenase, sickle cell anemia, beta-thalassemia trait, iron insufficiency or deficiency of carriers like GLUT1, AE1 are known to provide a certain degree of defense against a severe malarial course [230, 404]

Journal of the American Academy of Dermatology, 39 (4 Pt 1), 578589