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In rat embryonic myocytes with hypoxia/reoxygenation, trimetazidine pre-treatment mitigated the increase in oxidative stress proteins such as lactate dehydrogenase and ROS [169]

But its this origin that points to its primary area of study: cytoprotection, or the process of protecting cells from harm

Furthermore, it improved cognitive performance in scopolamine-induced memory impairment models and protected against serum and glucose deprivation injury in neuronal cells [69, 70]