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Interestingly, higher plasma levels of cystine indicated a high probability of response in patients with non-small cell lung cancer before and after treatment with the immune checkpoint inhibitor nivolumab, which targets programmed cell death protein 1

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A similar reaction could take place in the cellular environment, potentially mediated by other thiol-containing molecules such as glutathione (GSH), whose involvement is supported by subtle enhancement of ZZ1 potency in GSH-pretreated cells 45,46 (Extended Data Fig

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