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However, a negative regulatory interaction between Nrf2 and focadhesin (FOCAD), an adhesion plaque protein that enhances the sensitivity of NSCLC cells to ferroptosis under cysteine deprivation conditions, has been discovered [83]

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While these findings have not been linked to serious adverse outcomes so far, they highlight the need for long-term safety studies, which are currently underway in Phase 3 trials

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