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J Physiol Pharmacol 2012

But if study endpoints measure lasting structural brain changes, synaptic density, neuroprotection, or recovery from injury, the biological evidence supports intermittent administration

BPC 157 activated the Src-Cav-1-eNOS signal pathway and reduced eNOS/Cav-1 binding in vascular endothelial cells It is known that phosphorylation of Src can promote Cav-1 activation and subsequent endocytosis of tyrosine kinase receptors and eNOS activation 20,24