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Molecular docking studies reveal that hydrogen-bonded complexes formed between PPR and CUR further facilitate metabolic transport, raising toxicity concerns (32)

In light of the five-week timeline and lack of metabolic comorbidities with which our study was designed, is it likely that the origin of these changes was the ischemic myocardium itself

By replacing Alanine at position 8 with alpha-aminoisobutyric acid (Aib), the DPP-4 enzyme is unable to recognize the site, which signicantly enhances Semaglutide's resistance to degradation

doi: 10.4081/MONALDI.2021.1981 239 KocHCXiaoJLiuWLiYChenG

10.1074/jbc.M410818200