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This cascade of mitochondrial damage, along with the impaired ability of mitochondria to regulate calcium homeostasis and bioenergetics, leads to further neuronal injury and death [157]

It makes sense mechanistically, but the hard human clinical proof is limited

10.1053/jhep.2002.33201 126 SnykersS.HenkensT.De RopE.VinkenM.FraczekJ.De KockJ.et al (2009)

The former mechanism is preferred because RS scavengers can be consumed during the reaction and may generate toxic radicals derived from the chelators (Halliwell and Gutteridge, 2015)

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