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Metabolomic analyses of cerebrospinal fluid (CSF) from drug-resistant epilepsy patients show alterations in oxidative stress markers (e.g., lipid peroxidation products), suggesting possible ferroptotic activity (73, 74)
Third, despite formal risk of bias assessment, differences in trial duration, participant characteristics, and outcome reporting could still have influenced treatment effects
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In the context of GLP-1 co-activation (which prevents the blood sugar elevation that isolated glucagon would cause), the net result is increased energy expenditure without hyperglycemia
