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Dr John Eng and his colleagues discovered a peptide in the venom called exendin-4 , which shared structural similarities with human GLP-1 but possessed a crucial advantage: it was resistant to breakdown by DPP-4

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Specifically, this activation effect is mainly reflected in two aspects: it further activates the downstream mTOR molecules through the Akt-mTOR axis, directly promoting the synthesis of muscle proteins and inhibits the binding of the myostatin promoter, thereby reducing the expression of myostatin and ultimately alleviating muscle atrophy